parietal peritoneum
- 腹膜壁层;壁腹膜
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The Light Microscope and Scanning Electron Microscope Observation After Common Bile Duct Repair with Parietal Peritoneum and Jejunum Mucosal Flap
壁腹膜和空肠粘膜瓣修复胆管缺损后的光镜和扫描电镜观察
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Nodular thickening of parietal peritoneum ( 23 / 35 );
腹膜结节状增厚(23/35);
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Seroperitoneum was seen in 6 cases with thickened parietal peritoneum , greater omentum and lesser omentum in 3 cases .
6例(31.5%)腹腔积液;3例(15.8%)大网膜、肠系膜和壁层腹膜广泛结节样增厚。
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By transmission electron microscopy and freeze etching technique 15 human fetuses were utilized to study the ultrastructure of the mesothelial cells on the parietal peritoneum .
本文应用透射电镜和冷冻复型技术,对15例人胎腹膜壁层间皮细胞作了观察。
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There is guanophore distribution in the parietal peritoneum , which is compact and complete in the rest fish species except for the absence in transparent color crucian carp .
腹膜壁层分布有鸟粪素细胞,除水晶彩鲫缺失,其它品种致密完整。
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Results : The main CT manifestations of tuberculous peritonitis appeared as follows : ① Thickened parietal peritoneum in 29 cases ( 90.6 % ), including smooth in 26 and irregular in 3 , as well as obviously enhanced thickened parietal peritoneum in 18 ;
结果:①壁腹膜光滑、增厚29例(90.6%),其中均匀增厚26例,局部不规则增厚3例,18例增厚的腹膜有明显强化;
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Results The causes of congenital intra abdominal hernia were related to malrotation of intestine and abnormality of intestine fixation during embryogenesis , to erroneous juncture of visceral layer peritoneum with parietal peritoneum , and to partial degeneration and weakness of mesentery .
结果先天性腹内疝与胚胎发育期中肠的旋转与固定不正常及肠转位时脏层与壁层腹膜愈接不全或肠系膜的部分退化或薄弱有关;
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C57BL / 6 mouse were randomly divided into four groups : CTGF RNA interference-treated group , HK transfection group , peritoneal fibrosis group and normal control group . After corresponding treatments for 28 days , the parietal and visceral peritoneum was collected .
将C57BL/6小鼠随机分为CTGF干扰组、HK转染组、腹膜纤维化模型组和正常对照组4组,分别给予相应处理28天后,留取壁层和脏层腹膜组织。